eeeVis.this is what he's referencing
http://www.pnas.org/content/115/2/302
not outright wrong, but the entire post is just... bad
My biggest critique of this study is there's not a particular reason that male neuronal surface proteins would be exposed to the mother's immune system to
a. trigger initial immunity
b. trigger B-cell activation during the second pregnancy
further:
"The X-linked homolog, NLGN4X, might also play a role in the formation of sexual/romantic attractions, and antibodies raised by NLGN4Y might alter sexual brain development by cross-reacting with fetal NLGN4X, given its similarity to NLGN4Y"
would indicate the mother should face a fair chance of clonal deletion or anergy for any immune cells actually capable of targeting the gene, considering the epitopes are probably similar between the two
so idk man, p sus article imo
The science behind it might be different, who knows, but it doesn't deny the fact that such tendency exists.